{"id":2541,"date":"2022-01-19T17:42:45","date_gmt":"2022-01-19T22:42:45","guid":{"rendered":"https:\/\/pressbooks.bccampus.ca\/pathology\/?post_type=chapter&#038;p=2541"},"modified":"2025-01-13T14:11:34","modified_gmt":"2025-01-13T19:11:34","slug":"aki-post-test","status":"publish","type":"chapter","link":"https:\/\/pressbooks.bccampus.ca\/pathology\/chapter\/aki-post-test\/","title":{"raw":"Post-Test for Acute Kidney Injury","rendered":"Post-Test for Acute Kidney Injury"},"content":{"raw":"<div class=\"h5p\">[h5p id=\"114\"]<\/div>\r\n<div class=\"pdf\"><strong>1. Which of the following is an example of a prerenal cause of acute kidney injury?<\/strong>\r\n<ul>\r\n \t<li>Severe dehydration<\/li>\r\n \t<li>Acute glomerulonephritis<\/li>\r\n \t<li>Pelvic tumour blocking the flow out of the urethra<\/li>\r\n \t<li>Taking too many medications that hurt the kidney (nephrotoxic)<\/li>\r\n<\/ul>\r\n<strong>2. Finish the following sentence. The damage to the kidney in post-renal causes is generally caused by:<\/strong>\r\n<ul>\r\n \t<li>Inadequate blood flow to the kidney<\/li>\r\n \t<li>Toxic drugs<\/li>\r\n \t<li>The immune system<\/li>\r\n \t<li>Blockage of the drainage of urine<\/li>\r\n<\/ul>\r\n<strong>3. Which of the following signs is a result of protein loss in the urine from pathology that damages the glomerular filtration barrier?<\/strong>\r\n<ul>\r\n \t<li>Elevated blood pressure (hypertension)<\/li>\r\n \t<li>Edema (swelling)<\/li>\r\n \t<li>Increased amounts of dark urine<\/li>\r\n \t<li>Increased temperature (fever)<\/li>\r\n<\/ul>\r\n<strong>4. Which of the following is a histological sign of acute tubular injury?<\/strong>\r\n<ul>\r\n \t<li>Debris in the renal tubules<\/li>\r\n \t<li>Inflammatory cells filling the Bowmans space of the glomerulus<\/li>\r\n \t<li>Increase in brush border surface area in the PCT<\/li>\r\n \t<li>Increased number of neprhon cells (hyperplasia)<\/li>\r\n<\/ul>\r\n<strong>5. Fill in the blanks.<\/strong>\r\n\r\nAcute glomerulonephritis is an example of a _____ cause of AKI.\u00a0 It begins with damage to the _____, usually caused by the body's own _____ system.\u00a0 As a result, _____ can leak out of the blood and into the _____. This can result in inadequate osmotic gradient protein in the _____, which decreases fluid to stay in the tissues rather than be kept in the blood. Clinically, this can be seen as _____, most noticeably in the lower limbs and face.\r\n\r\n&nbsp;\r\n<div class=\"textbox\">\r\n<h2>Answer Key<\/h2>\r\n<ol>\r\n \t<li>severe dehydration<\/li>\r\n \t<li>Blockage of the drainage of urine<\/li>\r\n \t<li>Edema (swelling)<\/li>\r\n \t<li>Debris in the renal tubules<\/li>\r\n \t<li>intrarenal, glomerulus, immune, proteins, urine, blood, edema<\/li>\r\n<\/ol>\r\n<div><\/div>\r\n&nbsp;\r\n\r\n<\/div>\r\n<\/div>","rendered":"<div class=\"h5p\">\n<div id=\"h5p-114\">\n<div class=\"h5p-iframe-wrapper\"><iframe id=\"h5p-iframe-114\" class=\"h5p-iframe\" data-content-id=\"114\" style=\"height:1px\" src=\"about:blank\" frameBorder=\"0\" scrolling=\"no\" title=\"AKI Post-Test\"><\/iframe><\/div>\n<\/div>\n<\/div>\n<div class=\"pdf\"><strong>1. Which of the following is an example of a prerenal cause of acute kidney injury?<\/strong><\/p>\n<ul>\n<li>Severe dehydration<\/li>\n<li>Acute glomerulonephritis<\/li>\n<li>Pelvic tumour blocking the flow out of the urethra<\/li>\n<li>Taking too many medications that hurt the kidney (nephrotoxic)<\/li>\n<\/ul>\n<p><strong>2. Finish the following sentence. The damage to the kidney in post-renal causes is generally caused by:<\/strong><\/p>\n<ul>\n<li>Inadequate blood flow to the kidney<\/li>\n<li>Toxic drugs<\/li>\n<li>The immune system<\/li>\n<li>Blockage of the drainage of urine<\/li>\n<\/ul>\n<p><strong>3. Which of the following signs is a result of protein loss in the urine from pathology that damages the glomerular filtration barrier?<\/strong><\/p>\n<ul>\n<li>Elevated blood pressure (hypertension)<\/li>\n<li>Edema (swelling)<\/li>\n<li>Increased amounts of dark urine<\/li>\n<li>Increased temperature (fever)<\/li>\n<\/ul>\n<p><strong>4. Which of the following is a histological sign of acute tubular injury?<\/strong><\/p>\n<ul>\n<li>Debris in the renal tubules<\/li>\n<li>Inflammatory cells filling the Bowmans space of the glomerulus<\/li>\n<li>Increase in brush border surface area in the PCT<\/li>\n<li>Increased number of neprhon cells (hyperplasia)<\/li>\n<\/ul>\n<p><strong>5. Fill in the blanks.<\/strong><\/p>\n<p>Acute glomerulonephritis is an example of a _____ cause of AKI.\u00a0 It begins with damage to the _____, usually caused by the body&#8217;s own _____ system.\u00a0 As a result, _____ can leak out of the blood and into the _____. This can result in inadequate osmotic gradient protein in the _____, which decreases fluid to stay in the tissues rather than be kept in the blood. Clinically, this can be seen as _____, most noticeably in the lower limbs and face.<\/p>\n<p>&nbsp;<\/p>\n<div class=\"textbox\">\n<h2>Answer Key<\/h2>\n<ol>\n<li>severe dehydration<\/li>\n<li>Blockage of the drainage of urine<\/li>\n<li>Edema (swelling)<\/li>\n<li>Debris in the renal tubules<\/li>\n<li>intrarenal, glomerulus, immune, proteins, urine, blood, edema<\/li>\n<\/ol>\n<div><\/div>\n<p>&nbsp;<\/p>\n<\/div>\n<\/div>\n","protected":false},"author":1232,"menu_order":16,"template":"","meta":{"pb_show_title":"on","pb_short_title":"","pb_subtitle":"","pb_authors":["lyz-boyd","jen-2"],"pb_section_license":""},"chapter-type":[],"contributor":[59,64],"license":[],"class_list":["post-2541","chapter","type-chapter","status-publish","hentry","contributor-jen-2","contributor-lyz-boyd"],"part":2498,"_links":{"self":[{"href":"https:\/\/pressbooks.bccampus.ca\/pathology\/wp-json\/pressbooks\/v2\/chapters\/2541","targetHints":{"allow":["GET"]}}],"collection":[{"href":"https:\/\/pressbooks.bccampus.ca\/pathology\/wp-json\/pressbooks\/v2\/chapters"}],"about":[{"href":"https:\/\/pressbooks.bccampus.ca\/pathology\/wp-json\/wp\/v2\/types\/chapter"}],"author":[{"embeddable":true,"href":"https:\/\/pressbooks.bccampus.ca\/pathology\/wp-json\/wp\/v2\/users\/1232"}],"version-history":[{"count":8,"href":"https:\/\/pressbooks.bccampus.ca\/pathology\/wp-json\/pressbooks\/v2\/chapters\/2541\/revisions"}],"predecessor-version":[{"id":8247,"href":"https:\/\/pressbooks.bccampus.ca\/pathology\/wp-json\/pressbooks\/v2\/chapters\/2541\/revisions\/8247"}],"part":[{"href":"https:\/\/pressbooks.bccampus.ca\/pathology\/wp-json\/pressbooks\/v2\/parts\/2498"}],"metadata":[{"href":"https:\/\/pressbooks.bccampus.ca\/pathology\/wp-json\/pressbooks\/v2\/chapters\/2541\/metadata\/"}],"wp:attachment":[{"href":"https:\/\/pressbooks.bccampus.ca\/pathology\/wp-json\/wp\/v2\/media?parent=2541"}],"wp:term":[{"taxonomy":"chapter-type","embeddable":true,"href":"https:\/\/pressbooks.bccampus.ca\/pathology\/wp-json\/pressbooks\/v2\/chapter-type?post=2541"},{"taxonomy":"contributor","embeddable":true,"href":"https:\/\/pressbooks.bccampus.ca\/pathology\/wp-json\/wp\/v2\/contributor?post=2541"},{"taxonomy":"license","embeddable":true,"href":"https:\/\/pressbooks.bccampus.ca\/pathology\/wp-json\/wp\/v2\/license?post=2541"}],"curies":[{"name":"wp","href":"https:\/\/api.w.org\/{rel}","templated":true}]}}